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International Journal of Biology Sciences

Vol. 6, Issue 1, Part A (2024)

Effect study level of Interlukin (4, 8, 10) and Heamatological parameter in children infected with Enterobius vermicularis infection

Author(s):

Ahmed S Abed, Eman Hasani AL-Salami, Nadia Habeeb Sarhan and Dina AA Al-Roubaey

Abstract:

Background: Enterobius vermicularis, a human-pathogenic intestinal parasite that is a member of the nematode family and causes enterobiasis, is most commonly found in children. 
Objectives: The aim is to assess the magnitudes of interleukin (IL-4, IL-8, and IL-10) in children afflicted with pinworm. 
Supplies and Procedures: This research had 65 youngsters in all, 20 of whom were control individuals. Between the ages of one and fifteen, there were forty-nine girls and twenty-six men. The levels of IL-4, IL-8, and IL-10 were assessed by the use of an enzyme-linked immunosorbent assay.
Results: demonstrate that the infection rate is greater in females than in men, with the infection percentage in females reaching 60% and the infection percentage in males reaching 40%., However, a recent study revealed that those between the ages of 6 and 10 have the highest rate of pinworm parasite infection. Additionally to demonstrating a rise in (IL-8) concentration levels relative to the control group, the current investigation demonstrated a distinct decline in the majority of blood parameters in individuals with Enterobius vermicularis infection. In contrast to the group that was healthy However, a rise in eosinophilic blood cells was indicated by the data.
Conclusion: IL-4, IL-8, and IL-10 levels in the serum were rising in enterobiasis patients.
 

Pages: 38-41  |  236 Views  91 Downloads


International Journal of Biology Sciences
How to cite this article:
Ahmed S Abed, Eman Hasani AL-Salami, Nadia Habeeb Sarhan and Dina AA Al-Roubaey. Effect study level of Interlukin (4, 8, 10) and Heamatological parameter in children infected with Enterobius vermicularis infection. Int. J. Biol. Sci. 2024;6(1):38-41. DOI: 10.33545/26649926.2024.v6.i1a.184
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